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Increased NO flux can cause modification on tyrosine residues that induced the ubiquitin-proteasome pathway enhancing skeletal muscle apoptosis and atrophy [126,127]

As such, we expect the presented data to establish motivation for focused FBXO22 ligand discovery and degrader optimization efforts as well as for understanding a putative physiological relevance of reprogramming FBXO22

[DOI] [PMC free article] [PubMed] [Google Scholar] 147.Banach M., Serban C., Sahebkar A., Ursoniu S., Rysz J., Muntner P., Toth P.P., Jones S.R., Rizzo M., Glasser S.P., et al

Influence of Helicobacter pylori culture supernatant on the ecological balance of a dual-species oral biofilm
