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This chapter the role of redox state in apoptosis perturbation, exploring how the imbalance in redox status not only can directly responsible for tissue damage, but also promote development through the imbalance of immunological tolerance and the generation of neo-antigenic determinants from one side and of autoantibodies from the other

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The viability of N9 and N2a, as well as their recovery by cystine supplementation, were also evaluated in co-culture, which mimics the in-vivo condition to some extent
