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The literature reveals that Nrf2 plays a role in the development and progression of gliomas with isocitrate dehydrogenase mutations, further confirming an interplay between oxidative stress and isocitrate dehydrogenase mutations [26]

Moreover, the reaction rate of most antioxidants with ROS is much slower than that of antioxidant enzymes, and their actual antioxidant efficiency in vivo may be lower than expected [29, 30]

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