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In the LPS-induced AKI mouse model and the TKPTs treated with LPS, TMP195 improves renal tubule dilation, cell shedding, and renal tubule atrophy induced by LPS through inhibiting HDAC4 expression to exert anti-apoptotic and anti-inflammatory effects

Intravenous delivery results in immediate full-concentration exposure and typically the shortest apparent half-life because peak plasma levels degrade the fastest

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