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In addition, deletion of GLS1 genes from Myc-driven liver tumors upregulates several metabolic compensatory pathways, including glycolysis and aminotransferases 111

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However, when combination with HDAC4 (or HDAC5) with HDAC3, the transcriptional repression is facilitated via simultaneous binding of silencing mediator for retinoid and thyroid receptors (SMRT) and nuclear receptor corepressor (N-CoR) [63, 64]