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doi: 10.1093/jn/121.4.539 28

ACM Trans

We therefore hypothesised that the structurally flexible and less conserved D2 domain might be dispensable for maintaining S-layer assembly and engineered a mutant strain devoid of D2, named RD2 (producing SlpA RD2 )
This duality underscores the complexity of targeting LPL therapeutically, especially in individuals with diabetes or obesity, where both heart and vascular health are compromised