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& Bannister, W

The knockout of the HDAC3 gene does not affect the inflammatory regulator NF-B p65 cytoplasm-to-nucleus translocation and accumulation in nuclei, but enhances the acetylation of NF-B p65 at K122, decreases the transcriptional activity of NF-B p65, and reduces the expression of pro-inflammatory factors (TNF-, IL-1, and IL-6)

The research mechanism showed that GpS could increase AMPK activation and suppress adipogenesis by decreasing CCAAT/enhancerbinding protein (C/EBP), PPAR, sterol regulatory elementbinding protein1c (SREBP1c), PPAR coactivator1, fatty acid synthase, adipocyte protein 2, and sirtuin 1 (Lee et al., 2019)

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