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Description
Ligand binding to TLRs typically recruits the adaptor protein myeloid differentiation primary response 88 (MyD88), thereby triggering major pro-inflammatory signaling cascades, including the MAPK and nuclear factor kappa B (NF-B) pathways (259)

The predominant effects are increased GH gene transcription, enhanced GH synthesis, and amplification of secretory burst amplitude

Pittelkow 1981

[DOI] [PubMed] [Google Scholar] 320.Mai X.-M., Langhammer A., Camargo C.A., Chen Y
