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doi: 10.1007/s00380-005-0854-4

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We found that biotinylated sv6D and svC1 were poor substrates when assayed immediately after dissolving commercial lyophilized TGM2 in water containing 1 mM EDTA, pH 7.5, and 10 mM dithiothreitol (Figure 5a), but surprisingly, when the solution of TGM2 was stored over several days at 5 C in this medium, the enzyme lost over half of the activity with svL4 but gained the ability to use sv6D and, to a lesser extent, svC1 as substrates (Figure 5b)

CD38 deficiency protects heart from high fat diet-induced oxidative stress via activating Sirt3/FOXO3 pathway