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Description
Psychiatry 72 , 354360 (2012)
Finally, exendin4 significantly inhibited NF B (p65) translocation into the nucleus in MCF7 cells, a mechanism that was eliminated by the GLP1R antagonist exendin, proving again that this interaction occurs via GLP1R activation
21 Thereafter, it regulates learning, memory, 22 and neuron survival
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