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Table 3 shows the estimated decrease in A1c levels for different drugs in patients that are drug nave with an A1c of 8% and a weight of 90kg after 26 weeks of treatment

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They work by mimicking the action of the GLP-1 hormone, which is naturally produced in the gut

TCF7L2 is a possible regulator of GLP-1 receptor expression in human vascular cells Since there was correlation between the expression levels of GLP-1 receptor and TCF7L2 in the aorta in our experiments and it is well known that TCF7L2 is a transcriptional factor of GLP-1 receptor in pancreatic -cells, we hypothesized that TCF7L2 regulates GLP-1 receptor expression in human vascular cells as observed in -cells