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Description
The addition of energy expenditure enhancement via glucagon agonism addresses a limitation inherent to GLP-1-only approaches, which reduce energy intake but do not meaningfully increase energy output
Mood can also be impacted upon GLP-1 agonist cessation
Thresholds derived from Phase 2 dose-response data and tissue-specific glucagon receptor pharmacology
Many patients find it helpful to note the date, site, and any reactions in a medication diary or smartphone app
