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Description
Clinicians describe this effect as turning down the noise of addiction
Here, we find that semaglutide engages both G s and G q -dependent signalling pathways in Glp1r -expressing neurons in the area postrema (AP Glp1r ), the primary site of semaglutide action in the brain, and differentially regulates neuronal activation across distinct neuronal clusters
PMC 3687347
Additionally, they suppress glucagon secretion , slow gastric emptying, and promote satiety through central nervous system pathways, which collectively improve glycaemic control and facilitate weight loss
