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When HMGB1 is produced, it binds to TLR2, TLR4, and RAGE, initiating the HMGB1/TLR4/RAGE/NF-B cascade, which leads to the release of inflammatory cytokines and further aggregation of the secondary damage (Yang et al., 2005

In a spectromicroscopy study of iron NPs ranging from 80 nm to 6 nm, smaller particles demonstrated a marked increase in the initial rate of oxidation, attributed to their higher surface-to-volume ratio and enhanced surface reactivity

Future research should integrate traditional Chinese medical practices with modern medical studies to explore additional pathways between ferroptosis and UC