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Bmi1 overexpression in mesenchymal stem cells exerts antiaging and antiosteoporosis effects by inactivating p16/p19 signaling and inhibiting oxidative stress

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Instead, immature neutrophils rely on OXPHOS and FAO, with their energy production depending on degradation of lipid droplets through lipophagy with the resultant fatty acids directed to the TCA and OXPHOS (Riffelmacher et al., 2017)
