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In addition, glutathione (GSH) and N-acetylcysteine (NAC) restore intracellular redox equilibrium by replenishing GSH pools and activating AMPKNrf2HO-1 signaling, which promotes ROS detoxification and suppresses apoptotic cascades (89)

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The authors demonstrated that PSMA downregulation is due to the gain of CpG methylation, which was also associated with disease progression and reduced OS in mCRPC patients [175]

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