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Preclinical and in vitro work suggests that GHK-Cu can modulate collagen and glycosaminoglycan turnover, influence fibroblast and immune-cell behavior, and interact with pathways related to oxidative stress and tissue injury signaling

This PDF contains all fully annotated raw western blots analysed in this paper

Results Abundance of p62 in aneuploid cells scales with the amount of extra DNA We and others have previously shown that aneuploid cells accumulate autophagy and lysosomal markers on both transcriptome and proteome levels 8,14,15,16,19

Due to this interaction lactic acidemia impairs urate reabsorption and excretion while hyperuricemia results in impaired lactate excretion and reabsorption